参考文献/References:
[1]中国心血管健康与疾病报告编写组.中国心血管健康与疾病报告2019概要[J].中国循环杂志,2020,35(9):833-854.[2]Ling H,Gray CB,Zambon AC,et al.Ca2+/Calmodulin-dependent protein kinase Ⅱδmediates myocardial ischemia/reperfusion injury through nuclear factor-κB[J].Circ Res,2013,12(6):935-944.[3]Zhao L,Cheng G,Jin R,et al.Deletion of Interleukin-6 Attenuates Pressure Overload-Induced Left Ventricular Hypertrophy and Dysfunction[J].Circ Res,2016,118(12):1918-1929.[4]Yan J,Thomson JK,Zhao W,et al.Role of Stress Kinase JNK in Binge Alcohol-Evoked Atrial Arrhythmia.[J]Am Coll Cardiol,2018,71(13):1459-1470.[5]Grimm M,Ling H,Brown JH.Crossing signals:relationships between β-adrenergic stimulation and CaMKⅡ activation[J].Heart Rhythm,2011,8(8):1296-1298.[6]李胜胜,骞爱荣,罗明志,等.钙调蛋白激酶Ⅱ对细胞生理功能的调节作用[J].生命的化学,2011,31(1):95-101.[7]Bayer KU,Schulman H.Regulation of signal transduction by protein targeting: the case for CaMKII[J].Biochem Biophys Res Commun,2001,289(5):917-923.[8]Gray CB,Heller Brown J.CaMKⅡdelta subtypes:localization and function[J].Front Pharmacol,2014(5):15. [9]Srinivasan M,Edman CF,Schulman H.Alternative splicing introduces a nuclear localization signal that targets multifunctional CaM kinase to the nucleus[J].Cell Biol,1994,126(4):839-852.[10]贺文彬,张俊龙,于龙川,等.钙调素依赖性蛋白激酶Ⅱ的分子调节与突触可塑性[J].中国药理学通报,2014,30(7):897-901.[11]Zalcman G,Federman N,Romano A.CaMKⅡ Isoforms in Learning and Memory: Localization and Function[J].Front Mol Neurosci,2018(11):445.[12]Erickson JR.Mechanisms of CaMKⅡ Activation in the Heart[J].Front Pharmacol,2014(5):59.[13]Rosenberg OS,Deindl S,Sung RJ,et al.Structure of the autoinhibited kinase domain of CaMKⅡ and SAXS analysis of the holoenzyme[J].Cell,2005,123(5):849-860.[14]Rellos P,Pike AC,Niesen FH,et al.Structure of the CaMKⅡdelta/calmodulin complex reveals the molecular mechanism of CaMKⅡ kinase activation[J].PLoS Biol,2010,8(7):e1000426.[15]Erickson JR,He BJ,Grumbach IM,et al.CaMKⅡ in the cardiovascular system:sensing redox states[J].Physiol Rev,2011,91(3):889-915.[16]Erickson JR,Pereira L,Wang L,et al.Diabetic hyperglycaemia activates CaMKⅡ and arrhythmias by O-linked glycosylation[J].Nature,2013,502(7471):372-376.[17]Gutierrez DA,Fernandez-Tenorio M,Ogrodnik J,et al.NO-dependent CaMKⅡ activation during β-adrenergic stimulation of cardiac muscle[J].Cardiovasc Res,2013,100(3):392-401.[18]Jalan-Sakrikar N,Bartlett RK,Baucum AJ 2nd,et al.Substrate-selective and calcium-independent activation of CaMKⅡ by α-actinin[J].Biol Chem,2012,287(19):15275-15283.[19]Cheng H,Lederer WJ.Calcium sparks[J].Physiol Rev,2008,88(4):1491-1545.[20]Gordon JW,Shaw JA,Kirshenbaum LA.Multiple facets of NF-κB in the heart:to be or not to NF-κB[J].Circ Res,2011,108(9):1122-1132.[21]Martin TP,McCluskey C,Cunningham MR,et al.CaMKIIδ interacts directly with IKKβ and modulates NF-κB signalling in adult cardiac fibroblasts[J].Cell Signal,2018,51:166-175.[22]Gray CB,Suetomi T,Xiang S,et al.CaMKIIδ subtypes differentially regulate infarct formation following ex vivo myocardial ischemia/reperfusion through NF-κB and TNF-α[J].J Mol Cell Cardiol,2017,103:48-55. [23]Suetomi T,Willeford A,Brand CS,et al.Inflammation and NLRP3 Inflammasome Activation Initiated in Response to Pressure Overload by Ca2+/Calmodulin-Dependent Protein Kinase Ⅱ δ Signaling in Cardiomyocytes Are Essential for Adverse Cardiac Remodeling[J].Circulation,2018,138(22):2530-2544.[24]Willeford A,Suetomi T,Nickle A,et al.CaMKⅡδ-mediated inflammatory gene expression and inflammasome activation in cardiomyocytes initiate inflammation and induce fibrosis[J].JCI Insight,2018,3(12):e97054.[25]Kunisada K,Negoro S,Tone E,et al.Signal transducer and activator of transcription 3 in the heart transduces not only a hypertrophic signal but a protective signal against doxorubicin-induced cardiomyopathy[J].Proc Natl Acad Sci U S A,2000,97(1):315-319. [26]Hulsurkar M,Quick AP,Wehrens XH.STAT3: a link between CaMKⅡ-βIV-spectrin and maladaptive remodeling?[J].Clin Invest,2018,128(12):5219-5221. [27]Cai K,Chen H.MiR-625-5p Inhibits Cardiac Hypertrophy Through Targeting STAT3 and CaMKⅡ[J].Hum Gene Ther Clin Dev,2019,30(4):182-191. [28]Chang L,Karin M.Mammalian MAP kinase signalling cascades[J].Nature,2001,410(6824):37-40. [29]Yokota T,Wang Y.p38 MAP kinases in the heart[J].Gene,2016,575(2Pt2):369-376. [30]Palomeque J,Rueda OV,Sapia L,et al.Angiotensin Ⅱ-induced oxidative stress resets the Ca2+ dependence of Ca2+-calmodulin protein kinase Ⅱ and promotes a death pathway conserved across different species[J].Circ Res,2009,105(12):1204-1212. [31]周昌清,倪黎,晏小妮,等.美托洛尔下调CaMKⅡδC-p38通路对异丙肾上腺素诱导心力衰竭的保护机制[J].中国分子心脏病学杂志,2008(5):255-263.[32]Voskoboinik A,Prabhu S,Ling LH,et al.Alcohol and Atrial Fibrillation:A Sobering Review[J].J Am Coll Cardiol,2016,68(23):2567-2576. [33]Wang X,Lu Y,Xie B,et al.Chronic ethanol feeding potentiates Fas Jo2-induced hepatotoxicity: role of CYP2E1 and TNF-alpha and activation of JNK and P38 MAP kinase[J].Free Radic Biol Med,2009,47(5):518-528.[34]Huang Y,Li L,Li X,et al.Ginseng Compatibility Environment Attenuates Toxicity and Keeps Efficacy in Cor Pulmonale Treated by Fuzi Beimu Incompatibility Through the Coordinated Crosstalk of PKA and Epac Signaling Pathways[J].Front Pharmacol,2018,9:634. [35]Zhang X,Szeto C,Gao E,et al.Cardiotoxic and cardioprotective features of chronic β-adrenergic signaling[J].Circ Res,2013,112(3):498-509.[36]孙凤姣,张译丹,张密霞,等.β-AR/PKA/CaMK Ⅱ信号通路在阿霉素诱导大鼠心肌细胞凋亡中的作用[J].中国药理学通报,2017,33(3):360-365.[37]王赛,胡烨,包斯图.黄芩苷对扩张型心肌病大鼠心室重构、心室肌细胞凋亡及β1-AR/PKA/CaMKⅡ信号通路的影响[J].中国实验方剂学杂志,2018,24(9):140-144.
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